Search results for "cerebral vasospasm"

showing 10 items of 22 documents

Comparison of the anticonstrictor action of dihydropyridines (nimodipine and nicardipine) and Mg2+ in isolated human cerebral arteries.

1992

The isometric tension recorded from ring segments of branches of human middle cerebral artery was the parameter used to study the inhibition of spasmogen-induced contractions as model for cerebral vasospasm. Concentration-response curves to 5-hydroxytryptamine (10(-9)-3 x 10(-5) M) and prostaglandin F2 alpha (10(-7)-3 x 10(-5) M) were inhibited in Ca(2+)-free medium and in Ca(2+)-free medium to which EGTA (1 mM) had been added, respectively. Nimodipine (10(-7), 10(-5) M), nicardipine (10(-7), 10(-5) M) and Mg2+ (magnesium sulfate 10(-4), 10(-2) M) inhibited the 5-HT-elicited contractions, and this inhibition was similar for the highest concentrations tested. In contrast, nimodipine and nica…

AdultMaleSerotoninNicardipineCerebral arteriesProstaglandinPharmacologyIn Vitro TechniquesDinoprostchemistry.chemical_compoundNicardipineCerebral vasospasmmedicine.arterymedicineHumansMagnesiumNimodipineAgedPharmacologyAged 80 and overChemistryCerebral ArteriesMiddle AgedEGTAIschemic Attack TransientVasoconstrictionAnesthesiaMiddle cerebral arteryCirculatory systemCalciumFemaleNimodipinemedicine.drugEuropean journal of pharmacology
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Effect of intraarterial papaverine or nimodipine on vessel diameter in patients with cerebral vasospasm after subarachnoid hemorrhage.

2012

Papaverine (P) and nimodipine (N) are the most widely used vasodilators when angiographic and symptomatic vasospasm is present after subarachnoid aneurysmatic hemorrhage (SAH). Their effect is only short-lived and no direct comparisons have been undertaken to evaluate the action of both substances directly. We retrospectively assessed the effect of either P or N on angiographic diameter reduction and capillary blood flow.Fifteen SAH patients with secured aneurysms and cerebral vasospasm received intraarterial P, fifteen similar patients received N. As the primary endpoint, pre- and post-infusion arterial diameters and capillary blood flow were rated retrospectively on angiographies and comp…

AdultMaleSubarachnoid hemorrhageVasodilator AgentsIschemiaBrain IschemiaCerebral vasospasmModified Rankin ScalePapaverinemedicineHumansInfusions Intra-ArterialVasospasm Intracranialcardiovascular diseasesNimodipineAgedRetrospective StudiesAged 80 and overPapaverineAnalysis of Variancebusiness.industryVasospasmGeneral MedicineBlood flowCerebral ArteriesMiddle AgedSubarachnoid Hemorrhagemedicine.diseasenervous system diseasesCapillariesVasodilationAnesthesiaCerebrovascular CirculationSurgeryFemaleNimodipineNeurology (clinical)businessCarotid Artery Internalmedicine.drugBritish journal of neurosurgery
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Transcranial Doppler diagnosis of cerebral vasospasm following subarachnoid haemorrhage: correlation and analysis of results in relation to the age o…

1994

A retrospective analysis was undertaken to determine whether cerebral vasospasm following subarachnoid haemorrhage (SAH) correlates with the age of patients. For at least 3 weeks after bleeding 80 subjects underwent very close follow-up with clinical examination and transcranial Doppler records of the blood velocities within the basal cerebral arteries. Firstly a correlation between measured maximal mean blood flow velocities and age was made. Secondly, according to their age and the maximum of recorded mean velocities (v), the patients were divided into groups as follows: age 55 years or less, age more than 55 years; and maximum velocity v190 cm/s, 90 cm/sv2120 cm/s, 120 cm/sv3160 cm/s, v4…

AdultMaleUltrasonography Doppler TranscranialCerebral arteriesAneurysm RupturedDrug Administration ScheduleCerebral vasospasmMedicineHumanscardiovascular diseasesNeuroradiologyAgedRetrospective StudiesDose-Response Relationship Drugbusiness.industryVascular diseaseAge FactorsVasospasmIntracranial AneurysmLaser Doppler velocimetryMiddle AgedSubarachnoid Hemorrhagemedicine.diseasenervous system diseasesTranscranial Dopplermedicine.anatomical_structureIschemic Attack TransientAnesthesiaCerebrovascular CirculationSurgeryFemaleNimodipineNeurology (clinical)businessBlood Flow VelocityArteryFollow-Up StudiesActa neurochirurgica
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Endothelin: an endothelium-derived vasoactive peptide and its possible role in the pathogenesis of cerebral vasospasm

1991

The contractile response to endothelin has been examined in cerebral arteries from rats subjected to a prior subarachnoid haemorrhage (SAH) and compared with saline injected controls. Endothelin elicited strong concentration-dependent contraction of rat basilar artery segments. The response was slow in onset and long lasting. The endothelin-induced contraction was much stronger in the SAH compared to control animals. Our findings suggest a role of the peptide in the pathophysiology of cerebral vasospasm.

CEREBRAL ARTERYSettore MED/27 - NeurochirurgiaEndothelinscerebral vasospasm endothelinCEREBRAL ARTERY; ENDOTHELIN; SUBARACHNOID HEMORRHAGE; RATSMuscle Smooth VascularRATSIschemic Attack TransientBasilar ArteryCerebrovascular CirculationENDOTHELINAnimalsVasoconstrictor AgentsSUBARACHNOID HEMORRHAGEMuscle Contraction
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A Volumetric Method for Quantification of Cerebral Vasospasm in a Murine Model of Subarachnoid Hemorrhage

2018

Subarachnoid hemorrhage (SAH) is a subtype of hemorrhagic stroke. Cerebral vasospasm that occurs in the aftermath of the bleeding is an important factor determining patient outcome and is therefore frequently taken as a study endpoint. However, in small animal studies on SAH, quantification of cerebral vasospasm is a major challenge. Here, an ex vivo method is presented that allows quantification of volumes of entire vessel segments, which can be used as an objective measure to quantify cerebral vasospasm. In a first step, endovascular casting of the cerebral vasculature is performed using a radiopaque casting agent. Then, cross-sectional imaging data are acquired by micro computed tomograp…

MaleSubarachnoid hemorrhageGeneral Chemical EngineeringImaging dataGeneral Biochemistry Genetics and Molecular Biology030218 nuclear medicine & medical imagingMice03 medical and health sciencesCerebral circulation0302 clinical medicineCerebral vasospasmSmall animalAnimalsHumansVasospasm IntracranialMedicinecardiovascular diseasesStrokeGeneral Immunology and Microbiologybusiness.industryMicro computed tomographyGeneral NeuroscienceX-Ray MicrotomographySubarachnoid Hemorrhagemedicine.diseasenervous system diseasesMice Inbred C57BLDisease Models AnimalMurine modelcardiovascular systemNuclear medicinebusiness030217 neurology & neurosurgeryJournal of Visualized Experiments
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DOSE-RELATED EFFICACY OF A CONTINUOUS INTRACISTERNAL NIMODIPINE TREATMENT ON CEREBRAL VASOSPASM IN THE RAT DOUBLE SUBARACHNOID HEMORRHAGE MODEL

2009

Objective Intracisternal continuous therapy is a concept in the treatment of cerebral vasospasm after subarachnoid hemorrhage. The purpose of the current study was to investigate the effect of intracisternal nimodipine after induced vasospasm. Methods Sixty-five male Wistar rats were randomized into 4 groups: the control sham-operated group, the control subarachnoid hemorrhage-only group, and the treatment groups receiving 5 or 10 microL/hour of intracisternal nimodipine continuously for 5 days via subcutaneously implanted Alzet osmotic pumps (Durect Corp., Cupertino, CA). Vasospasm was analyzed 5 days later by means of digital subtraction angiography. Morphological examination of the brain…

MaleSubarachnoid hemorrhageRandom AllocationCerebral vasospasmmedicineAnimalsVasospasm IntracranialRats WistarNimodipineAntihypertensive AgentsDose-Response Relationship Drugmedicine.diagnostic_testbusiness.industryVasospasmIntracranial ArteryDigital subtraction angiographySubarachnoid Hemorrhagemedicine.diseaseCerebral AngiographyRatsDose–response relationshipAnesthesiaNimodipineSurgeryNeurology (clinical)medicine.symptomDrug ContaminationbusinessVasoconstrictionmedicine.drugNeurosurgery
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Vasoactive peptide urotensin II in plasma is associated with cerebral vasospasm after aneurysmal subarachnoid hemorrhage and constitutes a potential …

2019

National audience; OBJECTIVECerebral vasospasm (VS) is a severe complication of aneurysmal subarachnoid hemorrhage (SAH). Urotensin II (UII) is a potent vasoactive peptide activating the urotensin (UT) receptor, potentially involved in brain vascular pathologies. The authors hypothesized that UII/UT system antagonism with the UT receptor antagonist/biased ligand urantide may be associated with post-SAH VS. The objectives of this study were 2-fold: 1) to leverage an experimental mouse model of SAH with VS in order to study the effect of urotensinergic system antagonism on neurological outcome, and 2) to investigate the association between plasma UII level and symptomatic VS after SAH in huma…

SAPS II = Simplified Acute Physiology Score IIMCA = middle cerebral arteryAUC = area under the curvesubarachnoid hemorrhage[SDV]Life Sciences [q-bio]ICU = intensive care unitUT = urotensin (receptor)vascular disordersintensive care unitUII = urotensin IIcardiovascular diseaseshumanmouseWFNS = World Federation of Neurosurgical SocietiesEVD = external ventricular drainageACA = anterior cerebral arteryurotensin IInervous system diseasesSAH = subarachnoid hemorrhageSE = standard errorROC = receiver operating characteristic[SDV] Life Sciences [q-bio]cerebral vasospasmVS = vasospasmDCI = delayed cerebral ischemiaCSF = cerebrospinal fluidIRB = institutional review boardmRS = modified Rankin ScaleIQR = interquartile range
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Modulatory Role of Endothelial and Nonendothelial Nitric Oxide in 5-Hydroxytryptamine-induced Contraction in Cerebral Arteries after Subarachnoid Hem…

1996

OBJECTIVE : Endothelial dysfunction is claimed to play a role in the pathogenesis of delayed cerebral vasospasm after subarachnoid hemorrhage (SAH). We have examined the effect of experimental SAH on the modulatory action of endothelial and nonendothelial nitric oxide (NO) in the contractile response of goat middle cerebral artery to 5-hydroxytryptamine (5-HT). METHODS : We compared the 5-HT-induced contractile responses of cerebral arteries from control goats and from goats with SAH that had been experimentally induced 3 days earlier by delivery of autologous arterial blood into the subarachnoid space. Contractile responses were examined by recording the isometric tension in isolated cereb…

Serotoninmedicine.medical_specialtySubarachnoid hemorrhageEndotheliumCerebral arteriesNitric OxideCerebral vasospasmReference Valuesmedicine.arteryInternal medicinemedicineAnimalscardiovascular diseasesEndothelial dysfunctionVascular diseasebusiness.industryGoatsCerebral ArteriesSubarachnoid Hemorrhagemedicine.diseasenervous system diseasesmedicine.anatomical_structureEndocrinologyVasoconstrictionAnesthesiaMiddle cerebral arteryFemaleSurgeryEndothelium VascularNeurology (clinical)businessBlood vesselNeurosurgery
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Changes in the cerebrovascular effects of endothelin-1 and nicardipine after experimental subarachnoid hemorrhage.

1993

The role of endothelium-related factors in the pathogenesis of cerebral vasospasm after subarachnoid hemorrhage (SAH) has gained interest since the discovery of endothelin-1 (ET-1). We have examined, before and after SAH, the responsiveness of the cerebrovascular bed of the goat to ET-1, the sources of Ca2+ in ET-1-induced responses, and the ability of the Ca2+ entry blocker nicardipine to counteract them. Before SAH, injection of ET-1 into the cerebral circulation increased cerebrovascular resistance, thereby producing dose-dependent reductions in cerebral blood flow (CBF), which were prevented by nicardipine. In isolated middle cerebral arteries, ET-1 induced concentration-dependent contr…

Subarachnoid hemorrhageCerebral arteriesNicardipineBlood PressureCerebral circulationNicardipineCerebral vasospasmHeart RatemedicineAnimalscardiovascular diseasesDose-Response Relationship Drugbusiness.industryEndothelinsGoatsHemodynamicsBrainVasospasmSubarachnoid Hemorrhagemedicine.diseasenervous system diseasesmedicine.anatomical_structureCerebral blood flowVasoconstrictionAnesthesiaSurgeryFemaleVascular ResistanceNeurology (clinical)businessBlood Flow VelocityBlood vesselmedicine.drugNeurosurgery
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Effect of Intra-Arterial Infusion of Papaverine Hydrochloride on Brain Tissue Oxygen Pressure in the Management of Severe Vasospasm Following Aneurys…

2001

Despite considerable advances in diagnostic, surgical and interventional techniques, the overall outcome for patients with aneurysmal subarachnoid hemorrhage remains poor. Concerning perioperative course, cerebral ischaemia is one of the major factors causing secondary brain demage. The aim of all therapeutic interventions is the stabilization and maintance of cerebral blood flow and tissue oxygenation in order to prevent cerebral ischaemia and consecutively brain infarction [1].

Subarachnoid hemorrhagePapaverine Hydrochloridebusiness.industryVasospasmIntra arterial infusionPerioperativemedicine.diseaseIntensive care unitlaw.inventionCerebral vasospasmCerebral blood flowlawAnesthesiamedicinecardiovascular diseasesbusiness
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